摘 要 目的:探討多孔髓芯減壓支撐植骨并關節囊開窗治療早期股骨頭缺血性壞死的療效。方法:應用股骨頭多孔髓芯減壓支撐植骨并關節囊開窗治療早期股骨頭缺血性壞死患者46例(52髖)。行患髖多孔髓芯減壓支撐植骨并關節囊開窗術,臨床采用Harris評分系統評估,并分期X線檢查。結果:經平均隨訪18個月關節疼痛明顯緩解,功能明顯改善。影像學表現:股骨頭壞死區域有不同程度縮小,有部分股骨頭壞死發展緩慢,術前平均HARRS評分41.2,術后平均88.1。結論:多孔髓芯減壓支撐植骨并關節囊開窗可有效進行骨內及關節內減壓,改善內環境,減輕疼痛,阻止病情發展或延緩病情發展,該方法簡單有效,花費低,適合早期股骨頭缺血性壞死的患者。
關鍵詞 股骨頭缺血性壞死 髓芯減壓 支撐植骨 關節囊開窗
doi:10.3969/j.issn.1007—614x.2012.29.114
Abstract Objective:To Study the technique of medullary core decompression and implantation supporting bone and the capsular cutting window for the patients with avascular necrosis of femoral head at early stage by evaluate the clinical effect.Methods:Since October 2005 to June 2011,A retrospective study was made on 37 hips in 32 patients with avascular necrosis of femoral head at early stage by medullary core decompression and implantationation supporting bone and the capsular cutting window.The Harri’s hip rating score system and the.Results:of X—ray photos was used to evaluate the clinical efficacy of surgery.Results:All the patients were followed up for a mean time of 18months.The pain of the hips was decreased distinctly.The function of the hips was increased obviously.The X—ray photos showed that the zone of avascular necrosis of femoral head was decreased clearly.The average Harris score increased from preoperative 412 points to postoperative 881 points.Conclusion:It is sure and inexpensive that the effect of treatment avascular necrosis of femoral head by medullary core decompression and implantation supporting bone and the capsular cutting window.This method can be applied to patients with the avascular necrosis of femoral head at early stage.
Key words avascular necrosis of femoral head;medullary core decompression;implantationation of supporting bone;the capsular cutting window
股骨頭缺血性壞死是一種難治性疾病,給患者帶來極大的痛苦,非手術治療一般療效較差。2005年10月~2011年6月采用股骨頭多孔髓芯減壓支撐植骨并關節囊開窗治療早期股骨頭缺血性壞死,療效顯著,現報告如下。
資料與方法
本組患者46例,男35例,女11例;年齡28~55歲,平均38歲;病程6個月~2年,52髖受累,左側26例,右側16例,雙髖4例。按照ficat,s分期Ⅰ期9髖,ⅡA期23髖,ⅡB期17髖,Ⅲ期3髖。有長期飲酒史32例,激素服用史10例,不明原因4例。
治療方法:硬腰聯合麻醉,使用可透視手術床,術前30分鐘靜滴第3代頭孢菌素1次,平臥位,消毒范圍包括同側髂脊,首先取髂脊松質骨,骨刀向內掀起皮質骨,刮匙刮出松質骨,松質骨不沖洗時紗布包裹備用,皮質骨原位縫合,髂脊外形不受損,在C臂透視下確認壞死灶,自大粗隆下2cm呈三角形以2cm克氏針向壞死區打三枚克氏針,反復透視確認位置良好,以直徑4mm空心鉆擴髓,并用頸椎刻度刮匙盡可能刮除死骨,擴髓后用1:50肝素鈉鹽水借長穿刺針頭向孔內反復沖洗,松質骨向孔內植骨,植骨深度約3cm。從股骨頸前方切除約1.5cm×1.5cm關節囊,刮出肥厚滑膜,沖洗關節腔,注入玻璃酸鈉。術后應用抗生素1天,應用低分子肝素鈉,術后四天應用骨肽靜滴,早期借拐下床活動,應用拐杖2~3個月,臨床采用HARRIS評分系統評估,并定期X線或核磁共振檢查。
療效觀察:1髖關節疼痛功能臨床用HARRIS評分:HHS≥90分優,75~90分良,60~74.9分中,≤60分差,術后定期檢查壞死區縮小及新骨生長情況,X線形態學變化改善及MRI股骨頭壞死體積變小,視為聯合治療有效,應用SPSS12.0統計軟件分析,患髖疼痛緩解比較采用X2檢驗。
結 果
患髖疼痛緩解:患髖術后3天即有不同程度的減輕,不同分期患者術后5個月后,患髖疼痛明顯改善,隨時間延長各期患者疼痛均有明顯緩解(P<0.05),術后經隨訪6個月~3年,平均18個月。見表1。
X線及MRI檢查結果:術后5個月復查結果與手術前比較可見股骨頭壞死區有不同程度的縮小,可見少量新骨生成。
討 論
股骨頭缺血壞死是多種病因造成的股骨頭缺血和骨細胞壞死的病理過程,Wikes和Visscher認為1,骨髓組織壓力增高壓迫血管壁,增加血管外周阻力,降低靜脈回流,靜脈回流減低引起骨髓組織水腫,骨是一個密閉的腔室,組織水腫使髓腔內壓力進一步增加,形成惡性循環,導致骨缺血壞死,髓芯減壓使密閉的骨腔